Urolithin A improved diastolic function by 32% and reduced cardiac hypertrophy and fibrosis in obese hypertensive mice
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Summary
To confirm this, when urolithin A was administered to a mouse model of heart failure with obesity and hypertension, cardiac diastolic function improved by about 32%, cardiac hypertrophy decreased by 9.4%, and myocardial fibrosis decreased by about 42%. GIST announced on the 3rd that a research team led by Professor Chang-Myung Oh of the Department of Biomedical Engineering and a research team led by Professor Sang-Wook Park of Chonnam National University School of Dentistry have elucidated the mechanism by which 'Urolithin A', a gut microbiota-derived metabolite, restores damaged mitochondria and regulates gut microbiota metabolism to suppress the progression of 'heart failure with preserved ejection fraction (HFpEF)'. Editor's pick Recommended articles National pension good performance despite... 9.54 million in 'blind spots' Stock market boom tax revenue 'jackpot'...
Cross-referenced from 2 sources.
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To confirm this, when urolithin A was administered to a mouse model of heart failure with obesity and hypertension, cardiac diastolic function improved by about 32%, cardiac hypertrophy decreased by 9.4%, and myocardial fibrosis decreased by about 42%.
reliability low1/2 sourcesGIST announced on the 3rd that a research team led by Professor Chang-Myung Oh of the Department of Biomedical Engineering and a research team led by Professor Sang-Wook Park of Chonnam National University School of Dentistry have elucidated the mechanism by which 'Urolithin A', a gut microbiota-derived metabolite, restores damaged mitochondria and regulates gut microbiota metabolism to suppress the progression of 'heart failure with preserved ejection fraction (HFpEF)'.
reliability low1/2 sources
Reported detailssecondary facts, each attributed to its source
Editor's pick Recommended articles National pension good performance despite... 9.54 million in 'blind spots' Stock market boom tax revenue 'jackpot'...
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